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Clinically, T-cell signatures have been linked to the disease stage, frequency of progression, and non-motor symptoms, indicating their pathogenic importance [87]

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Newly identified entities such as DADA2 (deficiency of adenosine deaminase 2) and VEXAS (vacuoles, E1 enzyme, X-linked, autoinflammatory, somatic) syndrome, which are driven by genetic mutations, also exhibit vasculitic features and are associated with a high risk of AVTEs